T: Amyotrophic Lateral Sclerosis-Motor Neuron Disease
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| Preise | 2014 | 2015 | 2019 | 2021 |
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| Schnitt | Fr. 47.11 (€ 48.19)¹ | Fr. 55.18 (€ 56.44)¹ | Fr. 53.75 (€ 54.98)¹ | Fr. 50.83 (€ 51.99)¹ |
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1
T: Amyotrophic Lateral Sclerosis-Motor Neuron Disease (2014)
~EN PB NWISBN: 9783659614248 bzw. 3659614246, vermutlich in Englisch, LAP LAMBERT Academic Publishing, Taschenbuch, neu.
Lieferung aus: Österreich, Versandfertig in 3 - 6 Tagen.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Taschenbuch, 21.11.2014.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Taschenbuch, 21.11.2014.
2
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight (2014)
~EN PB NWISBN: 9783659614248 bzw. 3659614246, vermutlich in Englisch, LAP LAMBERT Academic Publishing, Taschenbuch, neu.
Lieferung aus: Österreich, Versandfertig in 4 - 6 Tagen.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Taschenbuch, 21.11.2014.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Taschenbuch, 21.11.2014.
3
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight
~EN NW ABISBN: 9783659614248 bzw. 3659614246, vermutlich in Englisch, neu, Hörbuch.
Lieferung aus: Schweiz, Lieferzeit: 2 Tage, zzgl. Versandkosten.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS.
4
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight - Amyotrophic Lateral Sclerosis: Molecular Neurobiology, Emerging Concepts and Novel Therapeutic Targets
~EN PB NWISBN: 9783659614248 bzw. 3659614246, vermutlich in Englisch, LAP Lambert Academic Publishing, Taschenbuch, neu.
Lieferung aus: Deutschland, Versandkostenfrei.
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight: Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig`s disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Englisch, Taschenbuch.
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight: Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig`s disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Englisch, Taschenbuch.
5
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight: Amyotrophic Lateral Sclerosis: Molecular Neurobiology, Emerging Concepts and Novel Therapeutic Targets (2014)
EN PB USISBN: 9783659614248 bzw. 3659614246, in Englisch, 128 Seiten, LAP LAMBERT Academic Publishing, Taschenbuch, gebraucht.
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Von Händler/Antiquariat, Wordery USA.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Paperback, Label: LAP LAMBERT Academic Publishing, LAP LAMBERT Academic Publishing, Produktgruppe: Book, Publiziert: 2014-10-28, Freigegeben: 2014-10-28, Studio: LAP LAMBERT Academic Publishing.
Von Händler/Antiquariat, Wordery USA.
Amyotrophic lateral sclerosis (ALS) also known as Lou Gehrig's disease, and rarely Charcot disease, is the most common form of motor neuron disease. It is typically characterized by degeneration of the motor neurons, and is usually fatal within a few years of onset. A subset of population suffering from ALS is due to inherited and genes mutation identified in familial cases have been involved in pathogenesis of ALS. Molecular mechanisms involved linked with SOD1-related ALS has revealed a complex array of interconnected pathological processes, including glutamate excitotoxicity, dysregulation of neurotrophic factors, axon guidance proteins, axonal transport defects, mitochondrial dysfunction, deficient protein quality control, and aberrant RNA processing. In humans, no therapy for ALS has ever been discovered that could extend lifespan more than a few additional months. The ALS Association has committed $99 million to find effective treatments and a cure for ALS. Our global research effort has helped increase the number of scientists working on ALS, advanced new discoveries and treatments, and has shed light on the complex genetic and environmental factors involved in ALS. Paperback, Label: LAP LAMBERT Academic Publishing, LAP LAMBERT Academic Publishing, Produktgruppe: Book, Publiziert: 2014-10-28, Freigegeben: 2014-10-28, Studio: LAP LAMBERT Academic Publishing.
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Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight (2014)
DE PB NWISBN: 9783659614248 bzw. 3659614246, in Deutsch, LAP Lambert Academic Publishing, Taschenbuch, neu.
Lieferung aus: Vereinigte Staaten von Amerika, zzgl. Versandkosten, Verandgebiet: STOCKNEW.
Von Händler/Antiquariat, Alibris, NV, Sparks, [RE:5].
Trade paperback.
Von Händler/Antiquariat, Alibris, NV, Sparks, [RE:5].
Trade paperback.
7
Amyotrophic Lateral Sclerosis-Motor Neuron Disease: An Insight: Amyotrophic Lateral Sclerosis: Molecular Neurobiology, Emerging Concepts and Novel Therapeutic Targets (2014)
EN PB NWISBN: 9783659614248 bzw. 3659614246, in Englisch, 128 Seiten, LAP LAMBERT Academic Publishing, Taschenbuch, neu.
Lieferung aus: Deutschland, Gewöhnlich versandfertig in 24 Stunden.
Von Händler/Antiquariat, Amazon.de.
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Von Händler/Antiquariat, Amazon.de.
Die Beschreibung dieses Angebotes ist von geringer Qualität oder in einer Fremdsprache. Trotzdem anzeigen
8
Amyotrophic Lateral Sclerosis-Motor Neuron Disease (2014)
~DE PB NWISBN: 9783659614248 bzw. 3659614246, vermutlich in Deutsch, Taschenbuch, neu.
Lieferung aus: Deutschland, Next Day, Versandkostenfrei.
Die Beschreibung dieses Angebotes ist von geringer Qualität oder in einer Fremdsprache. Trotzdem anzeigen
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